Chlorine is a strong oxidizing agent. There are several forms of exposure: industrial leaks, environmental re-leases occurring prirnarily in transport or water purification swimming pool-related events and houselhold-cleaning product misadventures. The toxic effect of inhaled chlorine gas on the respiratory tract has been known but there is insufficient evidence ta conclude that there is a chronic impairment of pulmonary function after acute or chronic exposure. Chlorine inhalation may manifest any of the full spectrum of pulmonary irritant effects, from minor mucosal response ta diffuse alveoler damage, may rapidly cause pulmonary edema and interstitial pne-umania, leading to acute hypoxemic respiratory failure. Pulmonary function tesis obtained following inhalation were most notable for airflow obstruction and air trapping. Follow up studies of World War 1 chlorine gas victims indicate that many of the survivors were subsequently disabled by asthma, chronic bronchitis and emphysema. fn aur cases, following chlorine gas inhalation widespread bronchial inflammation and hyperreactivity, and airway obstruction occurred. Airway obstruction resporıded to medicai treatment.