PULMONARY THROMBOEMBOLISM

It is the sudden occlusion of the pulmonary artery and its branches by an object such as thrombus air, fat, tumor parcast coming through the bloodstream (1). In the USA, about 630 thousand people are exposed to pulmonary embolism every year and about 200 thousand people are exposed to it. While the mortality rate is 8% in those who can be diagnosed and are treated, it approaches 30% in those who cannot be felt (2). Deep thrombosis was found in 33% of the cases (3,4). Pulmonary thromboembolism is a known complication from the beginning of the 19th pain. Previously, the thrombus was thought to be inside the artery, but later it was defined as embolic. In 1858, Wirchow first demonstrated that the so-called pulmonary thrombosis was an embolism, and he described a triad. Accordingly, local travnia at the wall of the vessel stays with the tendency of the blood to clot (5). These factors still maintain their validity in etiology. ETIOLOGY The reasons leading to pulmonary thromboembolism are mainly thrombi and lysis: fat, air, amnion tumor tissue, parasite eggs, foreign bodies such as catheters. Pulmonary artery embolism was found in 1 out of 3 cardiac autopsies on average, with mitral stenosis being in de-cornized cases. The right heart may be the source of thrombi in cases with atrial fibrillation or past myocardial infarction. In addition, factors that facilitate ventricular thrombosis are among the risk factors for pulmonary enriboli. Situations where the risk of pulmonary embolism is increased.