Makale Arama

We tried la search for the relationship between the location of myocardial infarction and the resulting complications in patients hokspitalized in the coronary care unit of the Teaching Hospital of Selçuk University with the diagnosis of myocardial infarction. Among fifty patients most of them suffered from anteriorly localized tnyocardial infarction which was followed by inferiorly localized myocardial infarction. The most cotnrnon complications were noticed iri anteriorly localized infarction whereas complications of inferiorly localized rnyocarclial infarction was of secondary frequency.

The part of population over 80 years old has increased in the last decades. Older population constitiutes major part of patients with cardiovascular diseases. The mortality, the morbidity and the frequency of acute coronary syndromes rise as the age of patients increases. The data related with the benefit of primary coronary intervention for old aged population is limited.Here we present 97 year old patient who applied with inferior myocardial infarction at the second hour of his chest pain and underwent to primary percutaneous intervention successfully. We wanted to discuss the current literature pertinent with our case.

In addition to the many research about organophosphate poisoning, the fact of acute myocardial infarction and respiratory failure that happen after organophosphate exposure is firstly reported. A 46 years old patient was taken into emergency with the plaint of dispne, chest pain and somnolence. By taking anamnesis, it is understood that these plaints are occured after eating cherry of which trees are disinfected with a pesticide (Dipterex SP 80 (%80 trichlorphon 0,0 dimethyl phosphonate). General situation was miserable, tachypnea and his consciosness was somnolence. The patient was immediately intubated and connected to the mechanical ventilator. There was sinus tachycardia in his electrocardiography and the elevation of pathologic Q and ST elevation in V1-4. Troponin was high in his blood test. With these indications, it was supposed that there was heart attack and respiratory failure depending on organophosphate poisoning on the patient. After the treatment of piralidoxime, acute myocardial infarction and respiratory failure, the patient was discharged from hospital for being angiography. Finally, acute myocardial infarction and respiratory failure which evolve out of organophosphate poisoning shouldn’t be ignored.

In order to stratificate the risk of aı-rhythmi• events (AE) such as sudden cardiac death, vent-ricular fibrillation, sustained vertricular tach-ycardia in the first year after myocardial infarction, 62 patients were evaluated with echocardiography, Holter monitorisation and high resolution signal averaged electrocarcliography (H1-RES ECG). Patients were divided into 2 subgroups according ta presence on absence of AE [AE (+): 5 (%8), AE (-): 57 (%92)]. Patients with AE [(mean left ventriculaı-eje•tion fr-action (LVEF): % 48±I 3.5 showed 5 (%100) late potentials (LP). 1 (%20) malign VA and 3 (%60) LV dysfunction with LVEF 5 %40, while the other patients with AE (-) (LVEF % 55±9.7) had 28 (49%) LP (+), 2(%3.5) malign VA ve 7 (%12) LVEF5._%40 (p<0.05, p>0.05, p<0.0I ). During the 1 year follow-up, LP (+) patients (n=33) who had 6 (18%) LVEF%40 and 3(%9) malign VA showed 5 (%15) sudden death, while LP (-) patients (n=29) who had 4 (13.8%) VEF5_%40 but no malign VA, showed nn sudden death (p>0.05, p>0.05, p<0.05). Patients with VA (n=14) who had 1 1(%78.5) LP (+) and 5.(%35.7) LVEF.40 showed 4 (%28.5) sud-den death in the first year after AMI, whereas pa-tients without VA (n=48) who had 22 (%45.8) LP ( +) and 5(%10) LVEF<%40 showed 1 (2%) sudden death (p<0.05, p<0.05, p<0.01 ). Patients with LVEF5_%40 (n=10), who had 6 (%60) LP (+) and 1 (%1 O) malign VA before hospital discharge evolved 3 (%30) sudden death, wheı-eas pa-tients with LVEF>%40 (n=52) who had 27 (%51 .9) LP (+) and 2 (%3.8) malign' VA before hospital dis•-harge, showed 2 (%3.8) sudden death in the one year follow-up period (p>0.05, p>0.05, p<0.01). In conclusion, in the risk stratification of arrhy-thmic events after AM!. alt of the thı-ee methods (ec-hocardiography, Holter, HI-RES ECG) are usefill where HI-RES ECG is more sensitive.

Coronary artery spasm is a special type of ischemic heart disease characterized by spontaneous episodes of chest pain accompanied by transitory ST segment elevations during the episodes. This syndrome may be associated with acute myocardial infarction, ventricular tachycardia or fibrillation, as well as with sudden death. Psychosocial stresses are also associated with an increased risk for acute myocardial infarction. We reported a 23 years old, male patient who applied to the emergency department for ongoing chest pain which had begun just after psychosocial trauma arising from heavy business tempo. He had neither ECG, nor cardiac enzym changes during admission, however acute MI with ST elevation occurred while following up period. There was no stenosis in coronary arteries on angiography. Given the typical symptoms, an ergonovine test was considered unnecessary. Administration of Ca-channel blocker and nitrates ameliorated his symptoms. The patient was prescribed a calcium channel blocker at discharge and has had no further chest pain. Finally, we thought that the cause of acute myocardial infarction for this young patient that occurred after vasospasm might be due to sympathetic hyperactivity and psychosocial stresses resulting from heavy work load conditions.

Aim: To present a congenital left ventricular diverticulum case a rare congenital abnormality and to review related literatüre. Case report: A 74-year-old male patient, admitted to our clinic with subacute myocardial infarction and detected the diverticulum in the inferior wall during left ventriculography. Conclusion: In our case, no treatment fort he diverticulum, small size of the diverticulum, and contraction as good as disappearing completely during ventricular systole.

Magnesium (Mg2 +) deficiency is likely to occur when acute myocardial infarction develops in patients with ischemic cardiomyopathy, diabetics, hypertensive patients treated with diuretics and diuretics. Potassium (K) deficiency may accompany Mg2 + deficiency. In 37 patients with acute myocardial infarction (AMI), serum K + and Mg2 + analyzes were performed and in 3 of 7 patients with hypomagnesemia, ventricular rhythm disturbances were detected (42.8%). In the group of 30 people with serum Mg2 + levels within normal limits, ventricular arrhythmia was detected in 4 (13.3%) patients.

Several studies have shown that infarct size is re-duced after trombolytic treatement in patients with acute myocardial infarction. The present study was pelformed to compare clinical and laboratory results in acute myocardial infarction patients following ei-ther intravenous thrombolysis or plasebo in our Co-ronary Care Unite. We peiformed intravenous strep-tokinase (n=46) within 6 hours from onset of chest hain. Total hospitalization days were not changed between patients with thrombolytic treatement and Placebo (10±4 gün vs 11 ±5 gün, p<0.304, respectively). CK-MB started significantly to change after 6 hours (176±138 mgldl, p<0.01). CPK, reached its peak level within 12 hours after thrombolytic treate-ment. SGOT, SGPT and LDII begun significantly change within 18 hours after thrombolytic treate-ment. Cholesterol, trigliserit, LDL were not changed significantly. But HDL increased significantly in pa-tients with thrombolytic treatement within 18 hours after thrombolytic treatment.

At'this study serum Mg levels of myocardial in-farction (n: 30) and paroxysınal supraventricular tachycardia (n: 30) vere coınpared with healthy con-trol subjects (n: 20). Mean serum Mg levels (mg1d1) vere 1.8 ± 0.3, 1.7 ± 0.4, 1.8 ± 0.2 respectively. There was no staüstically significant difference among groıps (p>0.05). Frequency of hypomagnese-mia was 10% (n: 2) for the control group, 13.3% (n: 4) for patients with acute ınıocardial infarction and 23.3% (n: 7) (p<0,05 ) for patients with supraventric-ular tachycardia. These results imply that hypomag-nesemia could Alay an important role at the genesis of suprave ntricular tachycardia.

Aim: Post-myocardial infarction ventricular septal defects (PMIVSD) are rare but have extremely high
mortality and morbidity. Mortality rates and risk factors vary in many studies. We aimed to evaluate
the mortality rates and risk factors of ventricular septal defects (PMIVSD) after myocardial infarction
performed in our center .
Patients and Methods: We retrospectively enrolled the patients who underwent surgery for PMIVSD in
our clinic from January 2006 to April 2020 with the age ≥ 18.
Results: A total of 9451 patients admitted to our center with acute myocardial infarction between 2006
and 2020 were examined. Twenty-eight patients operated for PMIVSD were included in the study. Our
PMIVSD rate was 0.296%. When we compare those survivors and non-survivors the only thing which
statistically significant was length of stay in hospital. In survivors it was 13 days when it 2 days in nonsurvivors
group (p<0.001). One patient in survivor group was st ayed in hospital for 107 days.
Conclusion: Timing of the surgery for PMIVSD patients is still challenging. Further strategies like
implanting LVAD or percutaneous devices and waiting for a while before the surgery will be the better
approach for PMIVSD patients.

In. this article, a case with interventricular sep-tum rupture which is one of the most serious coup-lications of acute myocardial infarction was int-roduced. The clinical situation of the patient with acute inferior myocard inlarction f1 /5 evaluated with the Doppler echocardiograph. Owing to this case differential diagnosis. diagnostics methods. prognosis and the best treatment of intervetricular septum rupture with acute myocardial infarction was discussed.

Cardiogenic shock is an emergency clinical state which is usually due to delile myocardial infarction and clzaracterized by hipoperfusion of vital organs. If the management isn'i on time- and isn't enough, patients with cardiogenic shock commonly die. Fortunately: IABC, by alleviating coroner circulation and left ventricular functions, can give the the chance ,fo• interventional cardiac procedures such as cardiac catheterization and revasvcularization.While generally accepted mortality rate is at least 60-70% in car-diogenic shock, it can be decreased with optimal IABC and revascularizarion. On the other hand, emergency PTCA in patients with acute myocardial infarction decreased the incidence of hospital mortality and cardiogenic shock. The design of this review is to investigate the clinical landnuırks• IABC and the efficacy of IABC, alone ör together with newer intervetztional cardiac procedures, on disease states that could cause cardiogenic shock.

This search was made in the coronary care unit of medical faculty of Selciik University between 1988 and 1991. 434 cases were therapied and ob-served. The aim of this research was to examine the factors which cause atrial _fibrillation and its effect on prognosis. While atrial fibrillation out of 434 cases occured in 53 patients (12%) in 41 of them (77%). AF oc-cured in Mil in the first 24 hours. Sinusal rythm re-versed spontaneously in 33 cases of (62%) in flue first two hours. In the old cases who had a preYeiosus myo-cardial infarction history it was observed that AF was more frequent and it was an important factor in the development of atrial infarction. The cardiac enzvm values were high in the cases who went in to AF as to the ones who dindn't into AF. In 24 of the patient didn't have, AF the cardiac failure were accompanying to the congestive car-diac failure. It was concluded that it was to the hemodynantic disorder which was caused by the width of necrosis area more than AF itself, despite increasing the hospital mortalty.